Long QT Syndrome Publications
Our Publications
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“Gene- and variant-specific efficacy of serum/glucocorticoid-regulated kinase 1 inhibition in long QT syndrome types 1 and 2,” Giannetti et al.
EP Europace, 2023;, euad094, doi.org/10.1093/europace/euad094
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“SGK1 Inhibition Attenuates the Action Potential Duration in Re-Engineered Heart Cell Models of Drug-Induced QT Prolongation,” Kim M, Sager PT et al.
Heart Rhythm (2023), doi: https://doi.org/10.1016/ j.hrthm.2022.12.036
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“SGK1 inhibition attenuated the action potential duration in patient- and genotype-specific re-engineered heart cells with congenital long QT syndrome,” Kim M, Sager PT et al.
Heart Rhythm February 15, 2023 DOI:https://doi.org/10.1016/j.hroo.2023.02.003
Reference Publications
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“Inhibition of serum & glucocorticoid regulated kinase-1 as novel therapy for cardiac arrhythmia disorders,” Bezzerides et al.
Sci Rep. 2017 Mar 23;7(1):346. doi: 10.1038/s41598-017-00413-3. PMID: 28336914; PMCID: PMC5428512
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“Risk Stratification in the Long-QT Syndrome,” Priori et al.
N Engl J Med. 2003 May 8;348(19):1866-74. doi: 10.1056/NEJMoa022147. PMID: 12736279
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“Effectiveness & Limitations of β-Blocker Therapy in Congenital Long-QT Syndrome,” Moss et al.
Circulation. 2000;101:616-623 doi: 10.1161/01.CIR.101.6.616
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“Modulating Effects of Age and Gender on the Clinical Course of Long QT Syndrome by Genotype,” Zareba et al.
JACC Vol. 42, No. 1, 2003 July 2, 2003:103–9 doi:10.1016/S0735-1097(03)00554-0
Atrial Fibrillation Publications
Our Publications
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SGK1-I protects mice from obesity-related AF. These data further highlight SGK1 and its inhibition as a compelling target in cardiac arrhythmogenesis.
Aneesh Bapat, Sabindra Pradhananga, Dinesh Srinivasan, Philip Sager and Patrick T Ellinor
Originally published 6 Nov 2023 Circulation. 2023;148:A15963
Reference Publications
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“Genetic inhibition of serum glucocorticoid kinase 1 prevents obesity-related atrial fibrillation,” Bapat…Das, Milan, et al.
JCI Insight. 2022 Oct 10;7(19):e160885. doi: 10.1172/jci.insight. 160885. PMID: 35998035.